Proposed Sequence of Structural Damage in Ankylosing Spondylitis
Category:
- Pathogenesis
How Does HLA-B27 Contribute to Susceptibility to Ankylosing Spondylitis?
Category:
- Pathogenesis
What Is the Risk of Developing Ankylosing Spondylitis if One of my Relatives Has the Disease?
Category:
- Pathogenesis
The Role of Genes in Ankylosing Spondylitis
Category:
- Pathogenesis
What Is the Role of HLA-B27 in the Inheritance of Ankylosing Spondylitis?
Category:
- Pathogenesis
Which Genes Influence Clinical Manifestations of Ankylosing Spondylitis?
Category:
- Pathogenesis
Three Different HLA-B27 Structures and Hypotheses as to How They Might Induce Disease Processes in AS
Category:
- Pathogenesis
Endoplasmic Reticulum Aminopeptidases (ERAP) and MHC Class-I Peptide Presentation
Category:
- Pathogenesis
Origin of Gut-Synovium Relation in SpA?
Category:
- Pathogenesis
Joint Expedition: Linking Gut Inflammation to Arthritis
Category:
- Pathogenesis
Murine Spondyloarthritis by Enhanced TNF mRNA Stability:the TNFΔARE Mouse Model
Category:
- Pathogenesis
Sacroiliitis in TNFΔARE Mice (Enhanced TNF mRNA Stability)
Category:
- Pathogenesis
A BMP* Antagonist (Noggin) Inhibits Ankylosis in an Animal Model of Ankylosing Enthesitis
Category:
- Pathogenesis
Active BMP Signaling in Achilles Tendon Enthesitis
Category:
- Pathogenesis
Influencing Ankylosis in Mice
Category:
- Pathogenesis
The Entheseal Stress Hypothesis
Category:
- Pathogenesis
IL-23 and Entheseal-Resident T Cells Promote Enthesitis and Osteoproliferation in Spondyloarthritis
Category:
- Pathogenesis
Systemic IL-23 Expression in vivo Induces Highly Specific Entheseal Inflammation
Category:
- Pathogenesis
Proposed Interaction between Inflammation and New Bone Formation in Spondyloarthritis
Category:
- Pathogenesis
New Bone Formation in AS
Category:
- Pathogenesis
Release of the TNF Brake Hypothesis in Ankylosing Spondylitis
Category:
- Pathogenesis
The Window of Opportunity for Disease Modification with Anti-Inflammatory Agents
Category:
- Pathogenesis
ERAP1 is Only Associated with HLA-B27 Positive AS
Category:
- Pathogenesis
Genetic Risk of Ankylosing Spondylitis in the Population
Category:
- Pathogenesis
The Relationship Between Spondyloarthritis, Reactive Arthritis (ReA), and Septic Arthritis
Category:
- Pathogenesis
What Known Genes are Involved in AS?
Category:
- Pathogenesis
AS Shares Genetic Associations More with ‘Seronegative’ than ‘Seropositive’ Diseases
Category:
- Pathogenesis
Pathology of Spondyloarthritis
Category:
- Pathogenesis
The Role of the Gut in Driving Joint Inflammation in Ankylosing Spondylitis
Category:
- Pathogenesis
The Role of the Gut in Driving Joint Inflammation in Ankylosing Spondylitis
Category:
- Pathogenesis
Potential Mechanisms by which ERAP Variants Operate to Influence Disease Risk in AS
Category:
- Pathogenesis
Tissue Specificity in the MHC-I-opathies
Category:
- Pathogenesis
Adaptive Immune Priming in the MCH-I-opathies
Category:
- Pathogenesis
The IL-23 Axis and Innate Lymphoid Cells in Target Tissues Affected by MHC-I-opathies
Category:
- Pathogenesis
Comparison of the Features of MHC‑I-opathies and Autoimmune Diseases
Category:
- Pathogenesis
Relationship between Gut Microbiota and Immune Function of the Host
Category:
- Pathogenesis
Multidirectional Model of Host–Microorganism Interplay
Category:
- Pathogenesis
Role of Innate Lymphoid Cells (ILCs) in Steady State, Non-Autoimmune and Autoimmune Chronic Inflammatory Diseases
Category:
- Pathogenesis
Human Innate Lymphoid Cell (ILC) Subsets
Category:
- Pathogenesis
New Immune Cells in Spondyloarthritis
Category:
- Pathogenesis
Enthesitis in Comparison with Synovitis
Category:
- Pathogenesis
Functional Model of Enthesitis
Category:
- Pathogenesis
Microanatomical Changes in Enthesitis
Category:
- Pathogenesis
Antigen Processing and Presentation: Potential Link to Ankylosing Spondylitis
Category:
- Pathogenesis
Type 3 Immunity in Ankylosing Spondylitis
Category:
- Pathogenesis